The first explanation of this phenomena came from an incarcerated physician. Although you can google it and information has been available since June 3, there has been no mention on the news. So here it is for those of you exposed, positive, or for future reference.
As written by the incarcerated physician:
“This coronavirus has an affinity for ACE2 receptors. These receptors line the endothelial cells of our blood vessels and are essential in the regulation of blood pressure. What researchers are finding out is that this COVID is more of a vascular disease than a pulmonary one.
Once the virus attaches to these cells, a cascade of events takes place whereby the body releases chemokines which in turn summon our immune cells to target and kill these virus infected cells. And once this happens, the effects of such lead to a condition called inflammation.
But here is where it gets interesting…..
Because of this inflammation, our bodies also produce micro thrombi, or small blood clots that travel throughout the blood stream and attach to various end organs. Now remember, these thrombi particles are rich in the COVID virus and now once they attach elsewhere the process of inflammation (swelling, fluid accumulation, tissue destruction, etc.) occurs wherever these thrombi land.
Doctors are finding out at autopsies that people are dying of other related complications of COVID besides the most common which is pulmonary. Why? Because these thrombi cause vessel damage and vessel blockages and this leads to tissue hypoxia or the inability of the tissues to receive oxygen from the blood.
As a result of this phenomena; patients are ending up on dialysis due to renal failure; respiratory distress syndrome requiring a ventilator; heart failure requiring cardiac catheterization; strokes in the brain and brain stem; liver failure and other GI complications including diarrhea and nausea and vomiting. And also the typical muscle aches, loss of smell, blue toes and blue fingers due to the terminal arteries of these digits having decreased blood flow and oxygenation.
And this is why physicians know that African Americans are 4 times more likely to die of this disease than whites and if you have one of the top major co-morbidities, you are 12 times more likely to die of COVID then a person who does not have one of these co-morbid conditions.
These conditions include diabetes, hypertension, congestive heart failure, obesity, chronic lung disease. Researchers are finding the problems lie in the small vessels within the lungs not getting enough oxygen because of the COVID infection at this level.
And now this gives the evidence of why we are seeing many patients slip into a fatal “second phase” of COVID……
Which means, you may have gotten over your initial symptoms like feelings of the flu, colds, and even pulmonary issues. But this second phase takes time to build up and this is what’s causing people to die 6-8 weeks after being initially diagnosed with COVID.
And currently there are a couple of drugs to help us:
- Statins which are primarily used for cholesterol reduction and also
- Dexamethesone – a steroid used to reduce inflammation
Researchers are now saying that “THIS VIRUS ACTS LIKE NO PATHOGEN HUMANITY HAS EVER SEEN”……”
I don’t know if there has been any treatment using the 81mg aspirin given to people to help thin the blood. In my opinion, if a person begins to show symptoms of Covid-19, it wouldn’t hurt to try. I wouldn’t recommend high doses of aspirin, because as an anti-pyretic, it works against the body’s temperature. And when you are dealing with a virus, you need the fever to help destroy the virus. But maybe the 81 mg dose wouldn’t affect the temperature that much, but would prevent the blood clot formation. Also, you don’t have to take it prophylactically, but just have it available and start if symptoms show. Even low-dose aspirin has side effects you don’t need if there isn’t a reason to take it. Remember this is not to be taken as medical advice, just information. For particular use, check with your physician.